Anti-KMT4 / Dot1L antibody
| Name | Anti-KMT4 / Dot1L antibody |
|---|---|
| Supplier | Abcam |
| Catalog | ab72454 |
| Prices | $390.00 |
| Sizes | 100 µl |
| Host | Rabbit |
| Clonality | Polyclonal |
| Isotype | IgG |
| Applications | IHC-P WB IP |
| Species Reactivities | Human, Monkey, Orangutan |
| Antigen | Synthetic peptide (Human) corresponding to a region between residues 1000 and 1050 of KMT4/ Dot1L |
| Description | Rabbit Polyclonal |
| Gene | DOT1L |
| Conjugate | Unconjugated |
| Supplier Page | Shop |
Product images
Immunohistochemistry (Formalin/PFA-fixed paraffin-embedded sections) analysis of human lung carcinoma tissue labelling KMT4 / Dot1L with ab72454 at 1/200 (1µg/ml). Detection: DAB.
All lanes : Anti-KMT4 / Dot1L antibody (ab72454) at 0.04 µg/mlLane 1 : HeLa whole cell lysate at 50 µgLane 2 : HeLa whole cell lysate at 15 µgLane 3 : HeLa whole cell lysate at 5 µgdeveloped using the ECL technique
HeLa whole cell lysate ( 1 mg for IP, 20% of IP loaded) with ab72454 at 3 µg/mg lysate for IP, followed by ab72454 at 1 µg/ml for WB. Detection: Chemiluminescence with an exposure time of 10 seconds.
Product References
Molecular mechanisms of selective estrogen receptor modulator activity in human - Molecular mechanisms of selective estrogen receptor modulator activity in human
Cirillo F, Nassa G, Tarallo R, Stellato C, De Filippo MR, Ambrosino C, Baumann M, Nyman TA, Weisz A. J Proteome Res. 2013 Jan 4;12(1):421-31.
The leukemogenic AF4-MLL fusion protein causes P-TEFb kinase activation and - The leukemogenic AF4-MLL fusion protein causes P-TEFb kinase activation and
Benedikt A, Baltruschat S, Scholz B, Bursen A, Arrey TN, Meyer B, Varagnolo L, Muller AM, Karas M, Dingermann T, Marschalek R. Leukemia. 2011 Jan;25(1):135-44.
Human Polymerase-Associated Factor complex (PAFc) connects the Super Elongation - Human Polymerase-Associated Factor complex (PAFc) connects the Super Elongation
He N, Chan CK, Sobhian B, Chou S, Xue Y, Liu M, Alber T, Benkirane M, Zhou Q. Proc Natl Acad Sci U S A. 2011 Sep 6;108(36):E636-45. doi: